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In vitro metabolism of the glucagon-like peptide-1 (GLP-1)-derived metabolites GLP-1 (9-36) amide and GLP-1 (28-36) amide in mouse and human hepatocytes

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We analyzed 34 randomized clinical trials comparing: Continuous positive airway pressure (CPAP) GLP-1 receptor agonists No active intervention Here is what the evidence shows: CPAP remains the most effective therapy for reducing AHI GLP-1 receptor agonists produce clinically meaningful BMI and HbA1c reductions Blood pressure effects were inconsistent Respiratory and metabolic domains behave differently This is not a competition between CPAP and GLP-1 therapy

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