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Compounds like Semax upregulate BDNF to promote synaptic plasticity and neuronal survival

These findings are also interesting from an IGF-1R targeting point of view because pharmacological modulations leading to an excess of MDM2 could be a manner to cause a selective IGF-1 inhibition and apoptosis in cancer cells

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the combined therapy shows better effects on blood glucose control and weight loss ( Statements Author contributions ZH: Writing original draft, Writing review and editing