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Description

This hypothesis is supported by our observation that ER-targeted LaccID was unable to catalyze DAB polymerization after cell fixation

Interestingly, a comparison of acute metabolic responses with the chronic profiles at the end of the treatment period strongly suggests that while the different GLP1-based agonists may share primary mechanisms of action, the durability and efficiency of their long-term metabolic responses differ, with clear superiority for the GLP1/E compound