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They may instead promote base excision repair and non-homologous end joining by increasing repair protein availability or by restricting ROS-mediated base damage

Animal studies involved naturally aged male C57BL/6 mice that were 20 months old and younger controls that were 3 and 13 months old

This comparison illustrates the gap between novel molecular mechanism discovery and validated clinical translation Dihexa represents frontier neuroscience with unresolved questions, while Cerebrolysin represents established but less mechanistically precise neurotrophic therapy

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