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Based on these findings, it was recognized for the first time that impairment of antioxidant defenses (vitamin E-deficiency) and enhanced levels of PUFAs (substrates for LPO) did not just amplify the injury mechanism but switched cell death signaling typically centered around mitochondrial dysfunction (in mice on normal diets) to one dominated by acute LPO [70]

Emerging roles of metallothioneins in beta cell pathophysiology: beyond and above metal homeostasis and antioxidant response

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Reduces oxidative stress Oxidative stress occurs when theres an imbalance between the production of free radicals and the bodys ability to fight them off