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Elucidating the mechanisms by which gut microbiota contribute to myocarditis pathogenesis may lead to the development of novel therapeutic approaches targeting the gut microbiota

This fact is relevant as transgenic mice overexpressing IGFBP-2 were resistant to developing obesity when fed with high fat diet [174], suggesting that this molecule could have a direct effect on preadipocyte differentiation

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(2) Extracellular vesicles (EVs)-mediated metastatic reprogramming through cargo remodeling (e.g., pro-inflammatory proteins) and dysregulation of oncogenic miRNAs