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In the model of phosphatidylcholine/phosphatidyserine (60:40) liposomes and hydrogen peroxide (H 2 O 2 ), Fe and Cu promote lipid peroxidation, interpreted as the consequence of the homolytic scission of H 2 O 2 and of endogenous hydroperoxides (ROOH) and of the generation of hydroxyl (HO ) and alcoxyl (RO ) radicals (Cadenas, 1989) depending strictly on the participation of Fe and Cu as redox-reactive metals