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But that peripheral-only action means it does not suppress appetite, does not change energy expenditure through central pathways, and does not produce the dramatic caloric deficits that GLP-1 agonists like semaglutide or tirzepatide create

In this episode of The Diary Of A CEO, Andrew Koutnik discusses the science and impact of the ketogenic diet, drawing from both research and his personal experience with type 1 diabetes and obesity

Goal: Studied for telomerase activation, telomere maintenance and circadian/melatonin regulation effects reported mostly in older Russian and preclinical work, not established in humans [5] [6]

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