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The combined quinone redox glutathione metabolism model can be used to simulate experimentally challenging scenarios such as potential redox cycling toxicity, while providing a platform from which quinone exposure and concentration toxicity experiments may be guided

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Vitamin D also upregulates PTH1R in keratinocytes, and treatment with the DNA methylation inhibitor 5-azacytidine (5-azaC) further augments Vitamin Dinduced CAMP expression [245]