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Semaglutide drives beta cell survival and proliferation through sustained GLP-1R-mediated PI3K-Akt anti-apoptotic signalling, PDX-1 transcription factor activation, and beta cell neogenesis biology , with the one-week pharmacokinetic window of Semaglutide enabling chronic beta cell GLP-1R trophic stimulation paradigms not achievable with shorter-acting incretin research compounds
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The in ovo administration of IGF-1 (100 ng/embryo, e12, into the albumen) resulted in increased body weight, muscle fiber diameter, muscle cross-sectional area, and the number of myofibers per unit area as well as the number of activated satellite cells and mitotic nuclei in leg and breast muscle of post-hatch ducklings
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