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While both compounds produce gastrointestinal side effects common to all incretin-based therapies, their severity and impact on treatment continuation differ markedly

Both animal and human studies demonstrate that LPS stimulate GLP-1 release via Toll-like receptors (TLRs)

Inhibition of monocyte adhesion to endothelial cells and attenuation of atherosclerotic lesion by a glucagon-like peptide-1 receptor agonist, exendin-4

In the LEAN trial, Liraglutide was shown to have a higher rates of NASH resolution and lower rates of fibrosis progression compared to placebo