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Despite the development of numerous drugs targeting specific pathological features of AD, such as amyloid-beta (A) deposition and neurofibrillary tangles, very few have been approved in the past 20 years, underscoring the limitations of single-target therapeutic approaches

Several studies have reported a prevalence of asthma ranging from 20 to 50% in individuals with homozygous (ZZ) and heterozygous (SZ) A1ATD ( It is hypothesized that augmentation therapy could improve inflammation, reduce bronchial hyperresponsiveness, and prevent the development of chronic airway changes in these patients

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