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(2010) Osteoarthritis and Cartilage Transgenic mouse model of accelerated OA treated with AOD 9604

In such circumstances, the driving force for the different cell ROS accumulating responses could be mainly contributing to the effects of MM induced by pre-LOsG treatment

doi: 10.4254/wjh.v7.i3.443 155 VisekrunaALuuMBiologyD

Previously, there is study revealed that promoting cellular intake of riboflavin by upregulating expression of SLC52A1 (a riboflavin transporter, also known as GPR172B/RFVT1) plays a crucial role in antisenescence via activating mitochondrial membrane potential and inhibiting the AMPK-p53 pathway [56]