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234 The nonsteroidal anti-inflammatory drug diclofenac can significantly reduce the protein expression level of NF-B p65 and the phosphorylation level of IB-, effectively protecting the structure and function of the intestinal barrier by suppressing the iNOS/NF-B pathway and activating the expression of HO-1

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Moreover, given that AD has been described as type 3 diabetes, with evidence suggesting peripheral metabolic dysfunction may play a role in disease initiation and progression (similar to the peripheral-to-central spread proposed in Parkinsons disease), blood transcriptomic analysis offers a relevant and practical window into the early disease mechanisms we sought to evaluate

doi: 10.2337/db16-0868 5 YuanSLarssonSC