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Through HDAC6 inhibition, the pathway promotes the removal of damaged cellular components, reducing oxidative stress and enhancing cellular repair mechanisms in retinal cells ( 2.2.3 Beyond the eye In renal tubular cells (Wang et al., 2021) and brain tissue in animal stroke model ( + -dependent deacetylase SIRT1, reducing mitochondrial dysfunction and oxidative stress by decreasing ROS production, thereby preventing apoptosis induced by lipotoxicity

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Research interest centres on its reported ability to activate telomerase, the enzyme that maintains the protective caps at the ends of chromosomes

To ensure the robustness of findings, sensitivity analyses were performed by excluding potentially influential observations identified through Cooks distance