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RAMP2 expression is shown to negatively modulate maximal inhibitory G i recruitment to the GcgR (40%), enhance maximal glucagon-stimulated cAMP production (2-fold) and potency (10-fold), improve oxyntomodulin-induced maximal cAMP production by 133%, and mediate GcgR binding promiscuity towards GLP-1R agonists when RAMP2 is not expressed [187]