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It has recently been shown that the appetite-suppressing effects of GLP-1RAs from the exendin group depend on pro-opiomelanocortin-producing neurons (POMC) and are mediated by the activation of arcuate POMC neurons [19]

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The intrinsic pathway is triggered by diverse intracellular stresses, such as DNA damage, metabolic perturbations, or endoplasmic reticulum stress, leading to mitochondrial outer membrane permeabilization (MOMP)