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Through its interaction with actin, Thymosin Beta 4 facilitates cellular movements and orchestrates the repair of damaged tissues, making it a promising therapeutic agent for various conditions

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The same principle applies to gut health

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Furthermore, Doxorubicin and Cyclophosphamide reduced synaptic plasticity via hindering expression of brain-derived neurotrophic factor, phosphorylated cyclase response element binding protein, synaptophysin, and postsynaptic density protein 95 whereas protein expression of such synaptic plasticity biomarkers was enhanced by L-carnitine treatment

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