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We examined YAP subcellular localization and, consistent with previous reports, observed a stiffness-mediated increase in nuclear YAP, with the strongest response at intermediate stiffness levels on COL IV and COL I, and on stiff FN-coated substrates (Fig

CD38 inhibition by apigenin ameliorates mitochondrial oxidative stress through restoration of the intracellular NAD+/NADH ratio and Sirt3 activity in renal tubular cells in diabetic rats

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