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In an animal model of stroke induced by carotid artery occlusion, thiamine was shown to reduce glutamate levels, which is a major factor in excitotoxicity and neuronal damage [15]

Second, confounding by indication is highly probable, as treatment allocation is driven by underlying metabolic severity and clinical characteristics that are themselves associated with dementia risk

In addition to GPX4, xCT, etc., DHODH on the surface of mitochondria exerts a protective effect on ferroptosis

Renal function should be monitored, as severe gastrointestinal adverse effects can lead to dehydration and acute kidney injury, particularly in patients with pre-existing renal impairment