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Cytosolic (BCATc) and mitochondrial (BCATm) forms of BCAT are encoded by BCAT1 and BCAT2 , respectively (22)

The low placental transfer could be caused by the release of Dipeptidyl peptidase IV positive syncytiotrophoblast-derived extracellular vesicles released by the human placenta which degrade GLP-1 in uterine blood (63), as well as to the high molecular weight of GLP-1 agonists (4 73 kDa)

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Shih-Jen Tsai, Semax, an analogue of adrenocorticotropin (410), is a potential agent for the treatment of attention-deficit hyperactivity disorder and Rett syndrome, Medical Hypotheses, Volume 68, Issue 5, 2007, Pages 1144-1146