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Studies in humans and animals with non-alcoholic fatty liver disease show that CD44 plays a key role in the inflammation and liver damage in NAFLD.[ref] Other genetic studies have looked at genes in pathways known to be involved in acetaminophen metabolism, including genes related to sulfation, glucuronidation, and glutathione production

When Cochrane reviewers grade an evidence base as very low quality despite a positive pooled effect, this is usually what they are reacting to: not that the benefit is fake, but that the conditions under which it was measured are the conditions that reliably make benefits look bigger than they are

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