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Other therapeutic approaches targeting fibroblast ferroptosis have also emerged: the in vitro and in vivo experiments demonstrate that platelet-rich plasma (PRP) promotes healing of type 2 diabetic mouse ulcers by inhibiting fibroblast ferroptosis and improving functional impairment [78, 80], providing a new strategy for diabetic ulcer treatment

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Quick Answer: The primary difference lies in bioavailability

However, after complexation with randomly substituted methyl -cyclodextrin the oral kinetics in rats became less variable due to rapid and complete absorption [32]