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this secondary damage releases more mtDAMPs, perpetuating the inflammatory cascade and amplifying the SIRS response beyond the initial ischemic insult

In chondrocytes from mice with IL-1-induced osteoarthritis, decreased protein expression of GPX4 and SLC7A11 and lipid ROS accumulation were observed, which inhibited the expression of type II collagen in chondrocytes and affected bone and joint health

In laboratory models, IGF-1 LR3 has been observed to initiate stronger receptor activation than the parent molecule, potentially resulting in more robust cellular responses

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