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It defined individual distinct RCD modalities, including intrinsic and extrinsic apoptosis, autophagy-dependent cell death, entotic cell death, ferroptosis, lysosome-dependent cell death, mitochondrial permeability transition-driven necrosis, mitotic death, necroptosis, NETotic cell death, parthanatos, and pyroptosis [4]

12,13 More recently there has been renewed interest on GHK-Cu for cognitive decline in aging

These exosomes inhibit MPP + -induced ferroptosis and lipid peroxidation, enhance HCMEC viability, wound healing, and migratory capacity, and promote cerebral angiogenesis