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Reads were aligned to the mouse GRCm38 genome using STAR v.2.5.2b

In IDH1 mutant glioblastoma, BCAT1 is transcriptionally suppressed because of the hypermethylation of three CpGs in the promoter, and this is also the consequence of IDH1 mutation-induced 2-hydroxyglutarate (2-HG) (Tonjes et al., 2013)

These neuroregeneration properties position Dihexa as a promising research compound for investigating brain repair mechanisms, developing treatments for degenerative neurological conditions, and understanding the potential for functional recovery following various forms of neural damage