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More recently, high concentrations of bile acids have been associated with increased GLP-1 concentrations and glycaemic improvements, which might be mediated through nuclear farnesoid-receptor X (FXR) and the G-protein-coupled bile acid-activated receptor (TGR5) on enteroendocrine L cells [14]

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Based on this evidence, it is recommended that long-term pharmacological management prioritize the optimization of treatment timing, formulation of personalized dosing regimens, and implementation of systematic efficacy monitoring and adverse reaction assessment protocols, to maximize therapeutic benefits while minimizing potential risks

IGF-1 LR3 doesn't initiate tumors but promotes existing ones through anti-apoptotic signaling