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Kidney diseases Pro-inflammatory effects In renal diseases, renal non-specific DAMPs such as HMGB1, histones, mtDNA, and uric acid activate PRRs to trigger innate immunity, which in turn mediates tubular inflammatory responses (Fig

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Wang X, Liang T, Jin A, Zhang C, Zhou J, Li M, et al

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Post-stroke fatigue as an indicator of underlying bioenergetics alterations

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