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Taken in conjunction with previous results, it suggests that while GLP1R and GR agonism in conjunction is able to produce large decreases in weight (Via the reduction of food intake and increased energy expenditure), but the absence of GIPR agonism produces a more unfavourable frequency of side-effects
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doi: 10.1210/en.2013-1934 34 ErenerS
33,34 JNK translocates to mitochondria, binds to SH3 homologyassociated Brutons tyrosine kinase (BTK) binding protein (Sab) on the mitochondrial membrane, and subsequently inactivates a proto-oncogene tyrosine-protein kinase (Src) in mitochondria, eventually leading to the dysfunction of the ETC and the increased release of reactive oxygen species (ROS)
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