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Patients with MDD exhibit KP dysregulation, often marked by an overproduction of quinolinic acid, an N-Methyl-D-aspartic acid receptor (NMDAR) agonist that drives excitotoxicity, alongside reduced production of kynurenic acid, an NMDAR antagonist that protects from excitotoxicity and has anti-inflammatory effects

Targeting or combined with ferroptosis pathway to regulate the cerebral environment is a very advisable treatment at present, and the inhibition of ferroptosis is likely to provide a new strategy for the prevention of neuron death induced by IS

This will help hydrate and nourish your skin, completing your skincare routine