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In mouse models of highfat dietinduced liver steatosis, it significantly inhibits hepatic NNMT activity, reduces NAM methylation, increases NAD+ and SAM levels, enhances mitochondrial fatty acid oxidation, reduces hepatic triglyceride and lipid accumulation, lowers lipotoxicity markers (e.g., malondialdehyde, transaminases), improves hepatocellular injury, and reverses steatosis

The best choice depends on how the peptide will be used

Loren Pickart isolated it in 1973 while studying age-related changes in liver cell function

However, anecdotal evidence is inherently unreliable because users who take AOD 9604 are typically also dieting, exercising, and sometimes stacking it with other compounds