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These results suggest that the cytotoxicity observed following treatment with MitoQ alone and in combination with DPI is not solely dependent on its ability to regulate mitochondrial superoxide production in MNA NB cells, which is in line with recent studies on MitoQ effects in other cancer cell types (31, 32)

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Therefore, the available literature on zonulin as a non-invasive marker for disease characterized by barrier dysfunction should be interpreted with extreme caution