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Human p53 is inhibited by glutathionylation of cysteines present in the proximal DNA-binding domain during oxidative stress

In laboratory research, GHK-Cu is used as a biochemical tool to study copper-dependent signaling pathways, extracellular matrix (ECM) regulation, redox balance, and transcriptional modulation in controlled in vitro and preclinical experimental models

they relate to endothelial function signalling pathways, including oxygen regulation and oxidative stress

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