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When oxygen availability is limited, glucose is the most metabolically efficient fuel, and during myocardial ischemia, the ratio of acetyl-CoA/CoA in mitochondria increases, leading to the activation of pyruvate dehydrogenase (PDH) kinase, which in turn phosphorylates PDH to inhibit its activity

In parallel, sHSPs, ranging from approximately 12 to 43 kDa, have emerged as critical modulators of protein aggregation and toxicity in the nervous system (Fig

CRISPR/Cas9 editing of glia maturation factor regulates mitochondrial dynamics by attenuation of the NRF2/HO-1 dependent ferritin activation in glial cells

It is contraindicated in patients with severe cardiovascular disease, uncontrolled hypertension, and in those with severe neurological disorders