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The ability to form sedentary communities or biofilms in anatomical sites, such as the middle ear, has also been shown to contribute to the in vivo persistence of NTHi (Garca-Cobos et al., 2014)

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Further exploration of these mechanisms and their associated signaling pathways has the potential to pave the way for the development of targeted therapies aimed at addressing diseases associated with impaired ferroptosis

A rising star involved in tumour immunity: Lactylation