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The secretome of EAT mirrors that of dysfunctional VAT characterized by tumor necrosis factor-alpha, interleukin-6, and their respective soluble receptors [123] So, EAT promoted the development and maintenance of AF through a pro-inflammatory and pro-fibrotic environment [124] leading to autonomic imbalance, connexin dysfunction, cell shortening, increase collagen deposition, and calcium overload [125, 126]

Ongoing care helps adjust dose, manage side effects, and track progress

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Insects 11:586 Zeng L, Li X, Liu J, Liu H, Xu H, Yang Z (2021) RNA-seq analysis reveals an essential role of the tyrosine metabolic pathway and inflammation in myopia-induced retinal degeneration in Guinea pigs