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Human pharmacokinetic data confirms sustained plasma glutathione levels supporting extended cellular antioxidant protection

Circulating Fe 3+ enters cells with the assistance of transferrin and subsequently converts to Fe 2+ ( 2+ can catalyze the generation of metabolically toxic reactive oxygen species and phospholipid peroxide through the Fenton reaction, leading to cell damage or death (Figure 4)

showed the possible role of nuclear GSTpi in the acquisition of resistance to anticancer drugs by cancer cells (66, 67), it is unclear how GSTP translocated from the cytoplasm to nucleus and if in the macrophage, cytosolic GSTP could also enter into the nucleus

& Samli, M