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Yin, X., Zhong, H., Ding, Y., Zhong, P., & Xu, S

The majority of these theories rely on the concepts of mtDNA copy number and mtDNA turnover

Chronic cerebral hypoperfusion induces microglial activation, polarized toward M1 (pro-inflammatory) phenotype, and the drug fingolimod has been shown, in an animal model, to attenuate such microglia-mediated neuroinflammation and to promote oligodendrocytogenesis by shifting microglia toward M2 subtype (217)

Can genetic testing clarify that