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Abstract Background Sepsis is typically hallmarked by high plasma (free) cortisol and suppressed cortisol breakdown, while plasma adrenocorticotropic hormone (ACTH) is not increased, referred to as ACTHcortisol dissociation. We hypothesized that sepsis acutely activates the hypothalamus to generate, via corticotropin-releasing hormone (CRH) and vasopressin (AVP), ACTH-induced hypercortisolemia
1.5108 cells/ml
Aluigi MG, De Flora S, D'Agostini F, Albini A, Fassina G: Antiapoptotic and antigenotoxic effects of N-acetylcysteine in human cells of endothelial origin
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