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Animal studies show substantial evidence across stroke models (full functional recovery in spatial memory, motor coordination, balance), Parkinsons models (protection of dopaminergic neurons, near-complete prevention of mortality), multiple sclerosis models (significantly less brain damage), and traumatic brain injury (reduced hemorrhage and edema)

The mechanism through which BPC-157 influences FAK-paxillin signaling, whether through integrin-associated signaling, NO-related modulation, VEGFR2-associated events, or another upstream process, has not been definitively established across all model systems

Thus, particularly following demonstration of the recovery of the myotendinous junction (dissection of quadriceps tendon from quadriceps muscle) [43], this study might provide a particular (cytoprotective) view and evidence of the muscle healing and function recovery with the cytoprotective stable gastric pentadecapeptide BPC 157 therapy [1,3,4,9,32,33,34]