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Notably, the oncogenic drivers prevalent in PDAC, such as mutant KRAS and loss of TP53 or SMAD4, intricately modulate key ferroptotic pathways, including the System Xc/GSH/GPX4 axis, the FSP1-CoQH system, and lipid metabolism networks, thereby dictating cellular susceptibility to ferroptotic stress

Next Steps for Your Journey: Avoid mixing medications without a providers explicit approval

Optimal results can take up to 16 weeks for some people

Patients transitioning from any other compounded GLP-1 platform should document their current medication, current weekly dose, duration at that dose, and clinical response in the Direct Meds intake questionnaire