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Multidrug resistance (MDR) in cancer

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16-OH-E1 is a proliferative estrogen, although less biologically active than estradiol

However, Aja and colleagues found no evidence for this hypothesis since in this model CPT-1 should inhibit feeding by increasing cytosolic fatty-acyl CoA levels while they actually showed the initial response of mice to a CPT-1 inhibitor was an increase in appetite [75]