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The accumulation of liver fat is the main pathogenic mechanism of NAFLD

Our data show that in pancreatic -cell lines, GRK2 interacts with the activated GLP-1R and that downregulation of GRK2 levels or pharmacological inhibition of its activity markedly impairs -arrestin2 recruitment to the agonist-stimulated receptor without apparent changes in Gs recruitment

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In addition, the large surface of proteins involved in the interaction among proteins is not covered by small molecules because their sizes are too small 124