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A loss-of-function study showed that Nrf2 knockout (Nrf2 (/) ) mice had more severe neurological deficits, brain edema, and neuronal cell apoptosis compared with Nrf2 (+/+) mice and were more susceptible to diseases related to oxidative stress, with higher expression of genes in the ER stress and ER stress-induced apoptotic pathways [60]

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doi:10.1016/j.ijsu.2010.02.007

The central role of Ca 2 as a secondary messenger underscores its versatility in encoding stress-specific information through distinct spatial and temporal patterns