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In cancer cells, heightened oxidative stress results from increased ROS production and/or compromised ROS-scavenging capacity [8]

An additional constraint could arise from the fact that while ferrostatin-1 has also previously shown efficacy in inhibiting ferroptosis in other human organoid models, such as bronchial [56] and gut crypt organoids [57], its utility is hindered by its solubility issues

This is further compounded by matrix metalloproteinase-9 (MMP-9) overexpression, which is induced by oxidative stress