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Mechanism of action when taken orally: Even though some oral glutathione is degraded in the gut, the resulting amino acids (cysteine, glycine, glutamate) serve as substrates for de novo glutathione synthesis in tissues

Some protocols discuss daily microgram ranges, but there is no universal dosage that fits every patient

During the short term (~6 h) after AKI has occurred, the glomerular filtration decreases sharply, the serum creatinine and blood urea nitrogen (BUN) increase, and the urine volume decreases, which results in variable kidney damage and eventually chronic kidney disease or terminal-stage renal disease (4,5)

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