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However, despite these beneficial effects, the persistence of the high-fat diet in the experimental model appears to have limited the ability of semaglutide to completely reverse the pathological activation of microglia

For instance, increased phosphorylation of APP -C-terminal fragment (CTF) Tyr682 inhibited the assembly and activity of V-ATPase by binding to the V0a1 subunit, resulting in elevated lysosomal pH and impaired degradation capacity