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Importantly, the demonstrated acute in vitro inhibitory effects of low concentrations of bumetanide on islet insulin secretion (Sandstrom, 1990) seem to stem mostly from inhibition of NKCC1, as its exclusive elimination from -cells precluded the effects of bumetanide ( However, experiments using islets of null mice lacking NKCC1 (NKCC1 KO ) gave unexpected results
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Comparison CompoundClassPathway / targetResearch focusFormat SemaxACTH-fragment peptideBDNF / NGF signallingNeurotrophic & cognitive researchVial SelankAnxiolytic peptide (Tuftsin analogue)GABA-BDNF interplayNeuropeptide / anxiolytic-model researchVial Semax + SelankPeptide blendCombined BDNF/NGF & GABA pathwaysComparative neuropeptide researchVial CerebrolysinMulti-peptide preparationNeurotrophic factor signallingNeurotrophic-pathway researchVial P21 (P021)Neurotrophic peptideTrkB receptorBDNF-mimetic signalling researchVial DihexaPeptidomimetic (small molecule)HGF / c-Met pathwaySynaptogenesis researchCapsules J-147Small moleculeMitochondrial / neurotrophic signallingNeuroprotective-model researchCapsules 9-Me-BCSmall molecule (beta-carboline)GDNF / dopaminergic pathwayNeurotrophic-model researchCapsules Methylene BlueSmall molecule (phenothiazine)Mitochondrial electron transport chainOxidative-stress & metabolism researchCapsules DSIPSleep-regulatory peptideDelta-wave / neuroendocrine axisSleep-signalling researchVial Every compound in this category is supplied as Research Use Only material with: Peer-reviewed literature underpinning this category
To view a copy of this licence, visit About this article Cite this article Carr, A., Samassa, F., Zhou, Z
