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Altogether, these results strongly support the hypothesis that KL1 enhances intracellular antibiotic efficacy by reducing oxidative and/or nitrosative stress in the host cell

As a triple-receptor agonist , its molecular structure must maintain a precise three-dimensional shape to bind effectively to GLP-1, GIP, and glucagon receptors

Importantly, the peptide does this without inducing tumor growth, suggesting it promotes healthy longevity