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Our assessment: mechanism is sound, preclinical support is strong, clinical translation has been disappointing
Control of mitochondrial and -cell function by SPHKAP-dependent GLP-1R signalling We next assessed the effect of ERMCS-localised GLP-1R-induced PKA signalling in the control of mitochondrial function in INS-1 832/3 cells
These increase production of a different hormone, GIP (glucose-dependent insulinotrpic polypeptide), but also impact the GLP-1 receptors
Time-restricted eating (limiting food intake to an 8-12 hour window) has shown some metabolic benefits in small studies, but mechanisms are not fully established and may involve multiple hormonal pathways
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